Ophthalmologists Warn that Fireworks-Related Injuries Can Cause Permanent Vision Loss

With Eye Injury Prevention Month approaching quickly and the use of fireworks increasing during the month of July, let’s all try to do out part in preventing unnecessary eye injuries.


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From Yahoo! News: SAN FRANCISCO, June 19, 2013 /PRNewswire-USNewswire/ — As the Fourth of July holiday approaches and Americans make plans to celebrate the stars and stripes with a little red glare from celebratory rockets, ranging from professional public fireworks displays to amateur impromptu backyard shows, the American Academy of Ophthalmology is urging the public to take important steps to prevent fireworks-related eye injuries. The Academy asks parents and other adults to exercise caution when handling fireworks themselves and to be especially diligent in managing and monitoring their use by children. …

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Five Activities To Engage Employees During National Safety Month – TFM

“National Safety Month is a reminder to organizations that safety is a continuous improvement process,”…


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By recognizing National Safety Month through events such as “Safety Awareness Day” or “Workplace Safety Week,” organizations can reduce workplace injuries and promote a culture of safety.

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The Word Myasthenia Gravis has Greek and Latin Root Meaning “Grave Muscular Weakness”

Myasthenia Gravis (pronounced My-as-theen-ee-a Grav-us) comes from the Greek and Latin words meaning “grave muscular weakness.” The most common form of MG is a chronic autoimmune neuromuscular disorder that is characterized by fluctuating weakness of the voluntary muscle groups. The prevalence of MG in the United States is estimated to be about 20/100,000 population. However, MG is probably under diagnosed and the prevalence may be higher. Myasthenia Gravis occurs in all races, both genders, and at any age. MG is not thought to be directly inherited nor is it contagious. It does occasionally occur in more than one member of the same family. The voluntary muscles of the entire body are controlled by nerve impulses that arise in the brain. These nerve impulses travel down the nerves to the place where the nerves meet the muscle fibers. Nerve fibers do not actually connect with muscle fibers. There is a space between the nerve ending and muscle fiber; this space is called the neuromuscular junction. When the nerve impulse originating in the brain arrives at the nerve ending, it releases a chemical called acetylcholine. Acetylcholine travels across the space to the muscle fiber side of the neuromuscular junction where it attaches to many receptor sites. The muscle contracts when enough of the receptor sites have been activated by the acetylcholine. In MG, there can be as much as an 80% reduction in the number of these receptor sites. The reduction in the number of receptor sites is caused by an antibody that destroys or blocks the receptor site. Antibodies are proteins that play an important role in the immune system. They are normally directed at foreign proteins called antigens that attack the body. Such foreign proteins include bacteria and viruses. Antibodies help the body to protect itself from these foreign proteins. For reasons not well understood, the immune system of the person with MG makes antibodies against the receptor sites of the neuromuscular junction. Abnormal antibodies can be measured in the blood of many people with MG. The antibodies destroy the receptor sites more rapidly than the body can replace them. Muscle weakness occurs when acetylcholine cannot activate enough receptor sites at the neuromuscular junction. – See more at:

http://www.myasthenia.org/WhatisMG.aspx#sthash.G4J7flBE.dpuf